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MYO Mosquito Repellent
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| In article <6NudnVHOS-EktjjZnZ2dnUVZ_o-dnZ2d@centurytel.net>,
unavailable@protected.email says...
 Did you know that Catnip has been scientifically proven to be 10x's more
effective than DEET? Here's a recipe for Mosquito Repellent and part of the
study:
http://www.budget101.com/MYOBugJuice.htm
More effective? If you mean by repelling mosquitos from one side of a
glass tube to another, yes it has shown to have that ability over DEET,
but then again DEET acts as a masking agent where they can't target you
while they are around you rather than a repelling agent that keeps them
totally away. I have never seen anything showing catnip protects from
bites over DEET.
--
Lar
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| Budget101.com 2006-07-01, 1:26 pm |
| > http://www.budget101.com/MYOBugJuice.htm
>
I have never seen anything showing catnip protects from
> bites over DEET.
Then you need to read the study at the bottom of the page (link above).
We've been using this spritz for over 2 years in our home and on our kids.
If you'd prefer to spray cancer causing deet on yourself, that's up to you!
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| gpsman 2006-07-01, 1:26 pm |
| Budget101.com wrote:
> I have never seen anything showing catnip protects from
>
> Then you need to read the study at the bottom of the page (link above).
Are you kidding? A study of 10 minutes of repellent effect? On glass?
That's what scientists call an "indication", not a "fact".
> We've been using this spritz for over 2 years in our home and on our kids.
> If you'd prefer to spray cancer causing deet on yourself, that's up to you!
Mmmm... the article says: "No animal or human tests are yet scheduled.
If testing proves safe for people..." Do ya think you might be jumping
the gun a little?
Deet is recommended to be primarily applied to clothing with small
amounts applied to the skin, and none to the skin of small children.
You're probably drenching your kids with an untested substance you know
nothing about and are assuming it's safe because it's "natural". Bad
idea, from my POV.
Your anecdotal evidence is irrelevant. I've talked to dozens of
homowners who think their electronic insect/rodent repellents work.
But, there I stand.
-----
- gpsman
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| In article <bqydnTeBy--epTjZnZ2dnUVZ_vGdnZ2d@centurytel.net>,
unavailable@protected.email says...
> http://www.budget101.com/MYOBugJuice.htm
>
I have never seen anything showing catnip protects from
> bites over DEET.
Then you need to read the study at the bottom of the page (link above).
We've been using this spritz for over 2 years in our home and on our kids.
The study mentions a repellency test that chases mosquitos from a
treated end of a glass tube to an untreated end of the same tube along
with stating that no human or animal tests (that would be for the biting
side rather than just repelling) are scheduled.
If you'd prefer to spray cancer causing deet on yourself, that's up to you!
You know something the rest of the world doesn't know? Can't seem to
find any information stating that DEET is a carcinogen.
--
Lar
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| Budget101.com 2006-07-02, 2:33 pm |
| > Can't seem to find any information stating that DEET is a carcinogen.<
FROM: DUKE university MEDICAL CENER
DEET (Chemical name: N,N-diethyl-meta-toluamide or
N,N-diethyl-3-methylbenzamide or Benzamide, N,N-diethyl-3-methyl-).
Studies have shown that DEET causes brain cell death and behavioral changes
in rats after frequent and prolonged use. This exposure causes neurons to
die in regions of the brain that control muscle movement, learning, memory,
and concentration. Rats treated with an average human dose of DEET (40 mg/kg
body weight) performed far worse when challenged with physical tasks
requiring muscle control, strength and coordination. These findings are
consistent with reported human symptoms following DEET's use by the military
in the Persian Gulf War.
With heavy exposure to DEET and other insecticides, humans may experience
memory loss, headache, weakness, fatigue, muscle and joint pain, tremors and
shortness of breath. These symptoms may not be evident until months or even
years after exposure. The most severe damage occurs when DEET is used
concurrently with other insecticides, such as permethrin, for prolonged and
frequent periods of time.
dukehealth.org.
------------------------------
[The Merck Index. 10th ed. Rahway, New Jersey: Merck Co., Inc., 1983.
412]**PEER REVIEWED**
CHILD EXPOSED TO PRODUCT CONTAINING M-DET EXPERIENCED DISORIENTATION,
STAGGERING GAIT, SLURRED SPEECH AND EPISODES CONSISTING OF STIFFENING INTO
SITTING POSITION, CRYING OUT, EXTENDING EXTREMITIES, FLEXING FINGERS AND
DORSIFLEXING TOES.
[Weast, R.C. (ed.). Handbook of Chemistry and Physics. 64th ed. Boca Raton,
Florida: CRC Press Inc., 1983-84.,p. II-346]**PEER REVIEWED**
A five-yr-old girl, sprayed with DEET nightly for three months, developed
headaches and slurred speech, progressing to athetosis, shaking, screaming,
and convulsions. She died 24 days after hospitalization. At autopsy the
brain showed generalized edema with intense congestion of meninges. There
was no demyelination and no evidence of meningitis. An 18-mo-old child who
ingested an unknown quantity of a liquid prepn of DET exhibited similar
signs and symptoms but eventually recovered.
[Gosselin, R.E., R.P. Smith, H.C. Hodge. Clinical Toxicology of Commercial
Products. 5th ed. Baltimore: XXXXXXXX and Wilkins, 1984.,p. II-346]**PEER
REVIEWED**
Seizures and acute behavior change developed in an 8 yr old girl following
exposure to Muskol and Off insect repellents. She recovered within 3 days
with supportive treatment; including anticonvulsant (phenytoin) medication.
The assumed toxic agent was N,N-diethyltoluamide.
[Roland EH et al; Can Med Assoc J 132 (2): 155-56 (1985)]**PEER REVIEWED**
One patient who was accidentally sprayed in the eye with OFF had immediate
smarting sensation which subsided rapidly when he flushed his eye with
water. Two hr later the only abnormality was fine gray stippling of the
corneal epithelium with tiny gray dots in the palpebral fissure. Vision at
that time was reduced from 20/15 to 20/20. The eye returned rapidly to
normal.
[Grant, W.M. Toxicology of the Eye. 3rd ed. Springfield, IL: Charles C.
Thomas Publisher, 1986. 338]**PEER REVIEWED**
This is a case report of a 6-yr-old girl who extensively used an insect
repellent containing N,N-diethyltoluamide (DET; DEET). The family history
and tests performed on the child indicated that the girl was deficient in
ornithine carbamoyl transferase (OCT). This deficiency was apparently
agitated by the extensive use of DET. Phthalyl alcohol was identified in the
child's liver at least 10 days after administration.
[Heick HMC et al; J Pediatr 97 (3): 471-3 (1980)]**PEER REVIEWED**
BULLOUS ERUPTIONS, SKIN NECROSIS, & PROLONGED DISABILITY WAS REPORTED IN
MILITARY PERSONNEL IN SOUTH VIETNAM. ALTHOUGH AN INSECT HAD BEEN PREVIOUSLY
THOUGHT WHOLLY THE CULPRIT, DIETHYL TOLUAMIDE, INSECT REPELLENT USED
PRODUCES SIMILAR ERUPTIONS. CAUTION ADVISED.
[LAMBERG SI, MULRENNAN JA; ARCH DERMATOL 100 (5): 582-6 (1969)]**PEER
REVIEWED**
A 42 year old woman with no prior atopic history touched a companion who had
just sprayed himself with repellent containing 52% deet. Generalized
pruritus rapidly developed and progressed to generalized angioedema. The
woman became nauseated and unconscious en route to hospital, where her blood
pressure was found to be 70/40 mm Hg. She responded to treatment with
epinephrine, diphenhydramine, and in corticosteroids. Periorbital edema
developed after another exposure to deet I week later. In a controlled
setting, a small amount of deet in isopropyl alcohol was applied to the
patient's forearm. Pruritus occurred in the treated area within 15 sec and
or progressed to localized urticaria despite immediate washing of the arm.
The patient was treated with epinephrine and diphenhydramine when she
reported pruritis of lips and the contralateral arm. She responded to
therapy, but the localized urticaria lasted for over 1 hr. Isopropyl alcohol
alone elicited non response.
[Hayes, W.J., Jr., E.R. Laws, Jr., (eds.). Handbook of Pesticide Toxicology.
Volume 3. Classes of Pesticides. New York, NY: Academic Press, Inc., 1991.
1503]**PEER REVIEWED**
A 35 year old woman presumed allergy reaction to insect when she had used
several repellents on frequent camping trips and noticed that a "red, raised
lesion" appeared about 30 min after application. Open patch testing on the
forearm with "pure" deet revealed, within 20 min, a macular erythema that
evolved into a wheal-and-flare response. Similar tests with dimethyl
phthalate and butopyronoxyl were negative. The response was passively
transferred, suggesting a possible immunologic mechanism. It was indicated
that this particular case of contact urticaria was of immediate-type
hypersensitivity (stage 1).
[Hayes, W.J., Jr., E.R. Laws, Jr., (eds.). Handbook of Pesticide Toxicology.
Volume 3. Classes of Pesticides. New York, NY: Academic Press, Inc., 1991.
1503]**PEER REVIEWED**
Several cases of a deet-associated toxic encephalopathy have been reported
in young females. A 3.5 year old girl suffered a bizarre illness after all
of a 180 ml aerosol can of deet had been used each evening for 2 weeks to
spray her and her night clothes and bedding. Because of this exposure and
because careful medical examination failed to suggest any other cause, the
possibility was considered that deet was the cause. However, it was pointed
out that, even if the child had absorbed all of the deet discharged from the
aerosol can, the dosage of active e ingredient would have been only 0.14
ml/kg/day, a level tolerated by animals. The signs were disorientation,
staggering gait, slurred speech, and episodes consisting of stiffening into
a sitting position, crying out, extending the extremities, flexing the
fingers, and dorsiflexing the toes. Therapy, which began 1 day after onset,
was symptomatic. Recovery was complete in 4 days.
[Hayes, W.J., Jr., E.R. Laws, Jr., (eds.). Handbook of Pesticide Toxicology.
Volume 3. Classes of Pesticides. New York, NY: Academic Press, Inc., 1991.
1503]**PEER REVIEWED**
A 30 year old man following self-medication with 75% deet for a papular,
truncal, erythematous rash that was later diagnosed as pityriasis rosea. It
was his recollection that he had used deet successfully to treat a similar
condition 4 years previously. Beginning 2 weeks prior to admission to the
hospital, he daily applied deet on one side of his body and entered a
homemade sauna for 60-90 min; he emerged from the sauna, treated the other
side of his body, and reentered the sauna for another 60-90 min. This
procedure was continued for 1 week. He was occasionally lethargic and
incoherent following the deet-sauna treatment. Four days prior to admission,
he developed marked personality changes that included delusions of grandeur
and verbal aggressivity. He became more irritable and belligerent and was
admitted to the hospital, where he required seclusion because of his violent
behavior. His condition worsened and was diagnosed as acute manic psychosis.
[Hayes, W.J., Jr., E.R. Laws, Jr., (eds.). Handbook of Pesticide Toxicology.
Volume 3. Classes of Pesticides. New York, NY: Academic Press, Inc., 1991.
1504]**PEER REVIEWED**
Serious adverse effects have occurred when used under tropical condition,
when it was applied to areas of skin that were occluded during sleep (mainly
the antecubital and popliteal fossae). Under these conditions, the skin
became red and tender, then exhibited blistering and erosion, leaving
painful weeping denuded areas that were slow to heal. Severe scarring
occasionally resulted from some of these severe reactions.
[U.S. Environmental Protection Agency/Office of Prevention, Pesticides, and
Toxic Substances. Reigart, J.R., Roberts, J.R. Recognition and Management of
Pesticide Poisonings. 5th ed. 1999. EPA Document No. EPA 735-R-98-003, and
available in electronic format at:
http://www.epa.gov/pesticides/safety/healthcare 80]**PEER REVIEWED**
Toxic encephalopathic reactions have apparently occurred in rare instances
following dermal application, mainly in children who were intensively
treated. The more frequently cause of systemic toxicity has been ingestion,
deliberate in adults, accidental in young children.
[U.S. Environmental Protection Agency/Office of Prevention, Pesticides, and
Toxic Substances. Reigart, J.R., Roberts, J.R. Recognition and Management of
Pesticide Poisonings. 5th ed. 1999. EPA Document No. EPA 735-R-98-003, and
available in electronic format at:
http://www.epa.gov/pesticides/safety/healthcare 80]**PEER REVIEWED**
Manifestation of toxic encephalopathy have been behavioral disorders
including headache, restlessness, irritability, ataxia, rapid loss of
consciousness, hypotension, and seizures. Some cases have shown flaccid
paralysis and areflexia. Deaths have occurred following very large doses.
Blood levels of DEET found in fatal systemic poisonings have ranged from 168
to 240 mg/l. Interpretation of DEET toxicity in some fatal cases has been
complicated by effects of simultaneously ingested ethanol, tranquilizers,
and other drugs. One well documented case of anaphylactic reaction to DEET
has been reported. One fatal case of encephalopathy in a child heterozygous
for ornithine carbamoyl transferase deficiency resembled Reyes syndrome, but
the postmortem appearance of the liver was not characteristic of the
syndrome.
[U.S. Environmental Protection Agency/Office of Prevention, Pesticides, and
Toxic Substances. Reigart, J.R., Roberts, J.R. Recognition and Management of
Pesticide Poisonings. 5th ed. 1999. EPA Document No. EPA 735-R-98-003, and
available in electronic format at:
http://www.epa.gov/pesticides/safety/healthcare 81]**PEER REVIEWED**
Application of deet to the face and arms of five volunteers daily for 5
consecutive days produced only slight irritation of the face and nose and
some desquamation about the nose. Similar changes, plus dryness of the face
and slight tingling sensation, occurred among those who received
applications for 3 consecutive days/week for 6 weeks, but all symptoms
disappeared during each 4-day period of rest.
[Hayes, Wayland J., Jr. Pesticides Studied in Man. Baltimore/London:
XXXXXXXX and Wilkins, 1982. 631]**PEER REVIEWED**
Skin, Eye and Respiratory Irritations:
IRRITANT TO ... MUCOUS MEMBRANES ...
[The Merck Index. 10th ed. Rahway, New Jersey: Merck Co., Inc., 1983.
412]**PEER REVIEWED**
Undiluted material is a moderate-to-severe irritant in the eye. Dermal
application of undiluted material and 50% solutions caused no primary
irritation in man ...
[Gosselin, R.E., R.P. Smith, H.C. Hodge. Clinical Toxicology of Commercial
Products. 5th ed. Baltimore: XXXXXXXX and Wilkins, 1984.,p. II-346]**PEER
REVIEWED**
THE UNDILUTED COMPD MAY IRRITATE MUCOUS MEMBRANES ...
[Worthing, C.R., S.B. Walker (eds.). The Pesticide Manual - A World
Compendium. 7th ed. Lavenham, Suffolk, Great Britain: The Lavenham Press
Limited, 1983. 194]**PEER REVIEWED**
Populations at Special Risk:
Discretion should be exercised in recommending DEET for persons who have
acne, psoriasis, an atopic predisposition, or other chronic skin condition.
It should not be applied to any skin area that is likely to be opposed to
another skin surface for a significant period of time (antecubital and
popliteal fossae, inguinal areas).
[U.S. Environmental Protection Agency/Office of Prevention, Pesticides, and
Toxic Substances. Reigart, J.R., Roberts, J.R. Recognition and Management of
Pesticide Poisonings. 5th ed. 1999. EPA Document No. EPA 735-R-98-003, and
available in electronic format at:
http://www.epa.gov/pesticides/safety/healthcare 81]**PEER REVIEWED**
Great caution should be exercised in using DEET on children.
[U.S. Environmental Protection Agency/Office of Prevention, Pesticides, and
Toxic Substances. Reigart, J.R., Roberts, J.R. Recognition and Management of
Pesticide Poisonings. 5th ed. 1999. EPA Document No. EPA 735-R-98-003, and
available in electronic format at:
http://www.epa.gov/pesticides/safety/healthcare 81]**PEER REVIEWED**
Probable Routes of Human Exposure:
NIOSH (NOES Survey 1981-1983) has statistically estimated that 9,275 workers
(458 of these are female) are potentially exposed to DEET in the USA(1).
Occupational exposure to DEET may occur through inhalation and dermal
contact with this compound at workplaces where DEET is produced or has been
and continues to be used(SRC). The general population may be exposed to DEET
via dermal contact with consumer products (insect repellent) containing
DEET(SRC). 30% of US population uses DEET annually as an insect
repellent(2). Approx 21% households use DEET annually; 19% of households use
it on household members; about 4% of households that have cats and/or dogs
use it on those pets(2).
[(1) NIOSH; National Occupational Exposure Survey (NOES) (1983) (2) USEPA;
Pesticide Reregistration Eligibility Decisions (REDs) Database on DEET
(134-62-3). USEPA-738/R-98-010. Available from the Database Query Page at
http://www.epa.gov/REDs/ as of Nov 30, 2000.]**PEER REVIEWED**
Average Daily Intake:
Daily exposure estimates for DEET are as follows: adult male, 12.10
mg/kg/day; adult female, 9.68 mg/kg/day; child, age 13-17, 21.05 mg/kg/day;
child, age 12 and under, 37.63 mg/kg/day(1).
[(1) USEPA; Pesticide Reregistration Eligibility Decisions (REDs) Database
on DEET (134-62-3). USEPA-738/R-98-010. Available from the Database Query
Page at http://www.epa.gov/REDs/ as of Nov 30, 2000.]**PEER REVIEWED**
http://www.pesticideinfo.org/refere...34-62-3hsdb.doc
Now, can you show me 1 single death caused by Catnip??
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| In article <i7adndtdUfrXXDrZnZ2dnUVZ_oydnZ2d@centurytel.net>,
unavailable@protected.email says...
> Can't seem to find any information stating that DEET is a carcinogen.<
FROM: DUKE university MEDICAL CENER
DEET (Chemical name: N,N-diethyl-meta-toluamide or
N,N-diethyl-3-methylbenzamide or Benzamide, N,N-diethyl-3-methyl-).
[Gosselin, R.E., R.P. Smith, H.C. Hodge. Clinical Toxicology of Commercial
And table salt causes kidney failure, peanut toxins can cause death,
many can not live with dairy products.... you stated that DEET causes
cancer...I asked what info you have show it to be a carcinogen. It is
easy to sell a product, in this case to steer traffic to your site, when
you tack on the "C" word to promote it yet doing so without it being the
case is this side of a con man. Sad part about it you probably play
enough on some people's emotions that we will see you spamming the
group, an information group, not a self promoting to generate revenue to
my web site group, shortly with another one of your miracle "links" to
drive revenue to your site.
--
Lar
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| Happybattles 2006-07-03, 1:25 pm |
| The fact is, nobody really knows anything about using catnip oil... at
all. Here's an example of an MSDS for Catnip Oil.
http://www.sciencelab.com/xMSDS-Cat...itation-9923338
Notice all the "unknown" areas? Plus... we all know what it does to
cats...
Here's some info from
http://chemistry.about.com/library/...r+mediterranean
============================
Nepetalactone is a terpene composed of two isoprene units, with a total
of ten carbons. Its chemical structure is similar to that of the
valepotriates derived from the herb valerian, which is a mild central
nervous system sedative (or stimulant to some persons).
[Hmm... central nervous system sedative... probably good for a buzz]
When cats smell catnip they exhibit a range of behaviors that may
include sniffing, licking and chewing the plant, head shaking, chin and
cheek rubbing, head rolling, and body rubbing. This psychosexual
reaction lasts for 5-15 minutes and cannot be evoked again for an hour
or more after exposure. Cats that react to nepetalactone differ in
their individual responses.
[Great for people with a... uh... cat fetish.]
The feline receptor for nepetalactone is in the vomeronasal organ,
located above the feline palate. The location of the vomeronasal organ
may explain why cats do not react from eating gelatin-enclosed capsules
of catnip. Nepetalactone must be inhaled for it to reach the receptors
in the vomeronasal organ. In cats, the effects of nepetalactone can be
moderated by several drugs acting upon the central and peripheral
nervous system, and by several environmental, physiological, and
psychological factors. The specific mechanism governing these behaviors
has not been described.
[Just as fun as feeding a dog beer.]
Catnip is an excellent sleep-inducing agent (as with valerian, in
certain individuals it acts as a stimulant). Both people and cats find
catnip to be emetic in large doses.
[Well... nobody's perfect.]
There is scientific evidence that catnip and nepetalactone may be
effective cockroach repellents.
[I'd rather kill them than repel them.]
Iowa State university researchers found nepetalactone to be 100x more
effective at repelling cockroaches than DEET, a common (and toxic)
insect repellent.
[Toxicity: the degree to which something is poisonous. Well,
everything is toxic to some degree. Heck, Di-hydro Oxide is
_EXTREMELY_ hazardous if you inhale it!]
Purified nepetalactone has also been shown to kill flies.
[So... again we're spraying ourselves with a pesticide.]
There is also evidence that nepetalactone may serve as an insect sex
pheromone in Hemiptera Aphidae (aphids) and a defense substance in
Orthoptera Phasmatidae (walking sticks).
[Mmm... so the mosquitoes stay away but you get aphids trying to hump
you... I guess mosquitoes are worse though...]
[The point is, there aren't any chemicals which are 100% safe for
anyone. Growing up in Arkansas, I used DEET every time I went fishing.
It helped protect me from mosquitoes, ticks, chiggers and the diseases
they carry. Peanut oil is tasty and useful but extremely hazardous to
some people. Shellfish make a great meal, so long as you aren't
allergic to them. Raspberries are a nice treat, but again, if you have
an allergy to them you're toast if you eat even one. Bee stings have
been shown to help reduce pain and inflammation in some people with
severe arthritis and other diseases but fatal in others.]
[I think this stuff is worth a shot for people who have shown a
sensitivity to DEET, but it will never replace DEET.]
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